Disorders of neuromuscular transmission other than myasthenia gravis
Identifieur interne : 003794 ( Main/Exploration ); précédent : 003793; suivant : 003795Disorders of neuromuscular transmission other than myasthenia gravis
Auteurs : Thomas R. Swift [États-Unis]Source :
- Muscle & Nerve [ 0148-639X ] ; 1981-07.
English descriptors
- Teeft :
- Abnormality, Acetylcholine, Acetylcholine receptors, Acetylcholine release, Achrs, Amyotrophic, Anesth analg, Antibiotic, Anticholinesterase, Anticholinesterase drugs, Anticholinesterase toxicity, Arch neurol, Botulinum, Botulinum toxin, Botulism, Cholinergic, Congenital, Corticosteroid, Decrement, Decremental, Decremental response, Defect, Direct effect, Direct effects, Disorder, Electromyographic, Electromyographic studies, Electromyography, Electrophysiologic, Electrophysiologic studies, Endplate, Endplate potentials, Envenomation, Etiology, Exhaustion, Facilitation, Gravis, Grune stratton, Guanidine, High rates, Human botulism, Junction, Lambert, Lithium, Lond, Magnesium, Magnesium intoxication, Mepp, Mepp amplitude, Minneap, Multiple sclerosis, Muscle action, Muscle action potentials, Muscle fiber, Muscle fibers, Muscle nerve, Muscle nerve juliaug, Muscle weakness, Myasthenia, Myasthenia gravis, Myasthenic, Myasthenic syndrome, Nerve, Nerve stimulation, Nerve terminal, Nerve terminals, Neurol, Neurol neurosurg psychiatry, Neurology, Neuromuscular, Neuromuscular block, Neuromuscular blockade, Neuromuscular junction, Neuromuscular junctions, Neuromuscular synapse, Neuromuscular transmission, Neuromuscular transmission disorders, Neuromuscular transmission disturbances, Neuropathy, Neurornuscular, Neurosurg, Physiol, Postactivation, Postactivation exhaustion, Postsynaptic, Postsynaptic mechanisms, Postsynaptically, Presynaptic, Ptosis, Quantum, Receptor, Releasable, Releasable store, Release sites, Repetitive, Repetitive nerve stimulation, Repetitive stimulation, Respiratory paralysis, Sclerosis, Several seconds, Skeletal muscle, Synapse, Synaptic, Synaptic vesicles, Syndrome, Tetanus, Toxin, Transmission, Transmission disturbances, Transmitter, Transmitter release, Venom, Vesicle, Voluntary contraction, West germany, Wound botulism.
Abstract
Disorders of neuromuscular transmission in humans are caused by a wide variety of agents including systemic diseases, drugs, environmental toxins, animal envenomation, cations, and hormones. Some are genetically determined. Many are of unknown etiology. All such disorders interfere with one or more events in the sequence whereby a nerve impulse excites a muscle action potential. In many disorders of neuromuscular transmission, abnormal fatigue occurs, and some cases respond dramatically to treatment. Investigation of the microphysiology, microanatomy, and pharmacology of both normal and diseased neuromuscular junctions has increased our knowledge of these disorders.
Url:
DOI: 10.1002/mus.880040410
Affiliations:
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Le document en format XML
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Swift</name><affiliation wicri:level="2"><country xml:lang="fr">États-Unis</country><placeName><region type="state">Géorgie (États-Unis)</region></placeName><wicri:cityArea>Department of Neurology, Medical College of Georgia, Augusta</wicri:cityArea></affiliation><affiliation wicri:level="2"><country xml:lang="fr">États-Unis</country><placeName><region type="state">Géorgie (États-Unis)</region></placeName><wicri:cityArea>Correspondence address: Department of Neurology, Medical College of Georgia, Augusta</wicri:cityArea></affiliation></author></analytic><monogr></monogr><series><title level="j" type="main">Muscle & Nerve</title><title level="j" type="alt">MUSCLE AND NERVE</title><idno type="ISSN">0148-639X</idno><idno type="eISSN">1097-4598</idno><imprint><biblScope unit="vol">4</biblScope><biblScope unit="issue">4</biblScope><biblScope unit="page" from="334">334</biblScope><biblScope unit="page" to="353">353</biblScope><biblScope unit="page-count">20</biblScope><publisher>Wiley Subscription Services, Inc., A Wiley Company</publisher><pubPlace>Hoboken</pubPlace><date type="published" when="1981-07">1981-07</date></imprint><idno type="ISSN">0148-639X</idno></series></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0148-639X</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="Teeft" xml:lang="en"><term>Abnormality</term><term>Acetylcholine</term><term>Acetylcholine receptors</term><term>Acetylcholine release</term><term>Achrs</term><term>Amyotrophic</term><term>Anesth analg</term><term>Antibiotic</term><term>Anticholinesterase</term><term>Anticholinesterase drugs</term><term>Anticholinesterase toxicity</term><term>Arch neurol</term><term>Botulinum</term><term>Botulinum toxin</term><term>Botulism</term><term>Cholinergic</term><term>Congenital</term><term>Corticosteroid</term><term>Decrement</term><term>Decremental</term><term>Decremental response</term><term>Defect</term><term>Direct effect</term><term>Direct effects</term><term>Disorder</term><term>Electromyographic</term><term>Electromyographic studies</term><term>Electromyography</term><term>Electrophysiologic</term><term>Electrophysiologic studies</term><term>Endplate</term><term>Endplate potentials</term><term>Envenomation</term><term>Etiology</term><term>Exhaustion</term><term>Facilitation</term><term>Gravis</term><term>Grune stratton</term><term>Guanidine</term><term>High rates</term><term>Human botulism</term><term>Junction</term><term>Lambert</term><term>Lithium</term><term>Lond</term><term>Magnesium</term><term>Magnesium intoxication</term><term>Mepp</term><term>Mepp amplitude</term><term>Minneap</term><term>Multiple sclerosis</term><term>Muscle action</term><term>Muscle action potentials</term><term>Muscle fiber</term><term>Muscle fibers</term><term>Muscle nerve</term><term>Muscle nerve juliaug</term><term>Muscle weakness</term><term>Myasthenia</term><term>Myasthenia gravis</term><term>Myasthenic</term><term>Myasthenic syndrome</term><term>Nerve</term><term>Nerve stimulation</term><term>Nerve terminal</term><term>Nerve terminals</term><term>Neurol</term><term>Neurol neurosurg psychiatry</term><term>Neurology</term><term>Neuromuscular</term><term>Neuromuscular block</term><term>Neuromuscular blockade</term><term>Neuromuscular junction</term><term>Neuromuscular junctions</term><term>Neuromuscular synapse</term><term>Neuromuscular transmission</term><term>Neuromuscular transmission disorders</term><term>Neuromuscular transmission disturbances</term><term>Neuropathy</term><term>Neurornuscular</term><term>Neurosurg</term><term>Physiol</term><term>Postactivation</term><term>Postactivation exhaustion</term><term>Postsynaptic</term><term>Postsynaptic mechanisms</term><term>Postsynaptically</term><term>Presynaptic</term><term>Ptosis</term><term>Quantum</term><term>Receptor</term><term>Releasable</term><term>Releasable store</term><term>Release sites</term><term>Repetitive</term><term>Repetitive nerve stimulation</term><term>Repetitive stimulation</term><term>Respiratory paralysis</term><term>Sclerosis</term><term>Several seconds</term><term>Skeletal muscle</term><term>Synapse</term><term>Synaptic</term><term>Synaptic vesicles</term><term>Syndrome</term><term>Tetanus</term><term>Toxin</term><term>Transmission</term><term>Transmission disturbances</term><term>Transmitter</term><term>Transmitter release</term><term>Venom</term><term>Vesicle</term><term>Voluntary contraction</term><term>West germany</term><term>Wound botulism</term></keywords></textClass></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Disorders of neuromuscular transmission in humans are caused by a wide variety of agents including systemic diseases, drugs, environmental toxins, animal envenomation, cations, and hormones. Some are genetically determined. Many are of unknown etiology. All such disorders interfere with one or more events in the sequence whereby a nerve impulse excites a muscle action potential. In many disorders of neuromuscular transmission, abnormal fatigue occurs, and some cases respond dramatically to treatment. Investigation of the microphysiology, microanatomy, and pharmacology of both normal and diseased neuromuscular junctions has increased our knowledge of these disorders.</div></front></TEI><affiliations><list><country><li>États-Unis</li></country><region><li>Géorgie (États-Unis)</li></region></list><tree><country name="États-Unis"><region name="Géorgie (États-Unis)"><name sortKey="Swift, Thomas R" sort="Swift, Thomas R" uniqKey="Swift T" first="Thomas R." last="Swift">Thomas R. Swift</name></region><name sortKey="Swift, Thomas R" sort="Swift, Thomas R" uniqKey="Swift T" first="Thomas R." last="Swift">Thomas R. 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